This page provides guidance on bodybuilding and weight loss from a metabolic-friendly perspective, inspired by Ray Peat's principles. The approach emphasizes minimizing stress, supporting anabolic processes, and maintaining a high metabolic rate through non-destructive resistance training, proper fueling, and sustainable habits. Bodybuilding focuses on building quality muscle mass to enhance strength, metabolism, and overall health, while weight loss prioritizes slow, toxin-minimizing fat reduction without compromising muscle or thyroid function. Many principles overlap, such as resistance training's benefits for both goals, and adaptations for fat loss are highlighted in the second section.
Train 2 to 3 times a week, concentric-dominant, and stop before fatigue rather than pushing to failure. A one-hour outdoor walk with friends does more for stress and enjoyment than an intense gym session. Keep goals enjoyable and sustainable, not punishing.
Concentric resistance training has an anabolic effect on the whole body. Sprinting is probably o.k. Endurance exercise is the worst. I don't think martial arts are necessarily too stressful.
The heart contracts against a load and relaxes without a load. Heartmitochondria do not change during ordinary aging. Concentric-dominant movements with relaxed recovery phases follow this pattern.[2]
Twelve weeks of mental training alone (15 min/day, 5 days/week; little-finger abduction or elbow-flexion imagery; 30 volunteers) raised finger-abduction strength 35% and elbow-flexion strength 13.5%, versus 53% with physical finger training and no change in untrained controls; gains tracked higher EEG-derived cortical potential related to voluntary contraction control.[4]
One moderate lifting session (about 42 min; two sets of 10 on six machine and dumbbell lifts at 65–75% of one-repetition maximum; 121 adults aged 18–50) sped executive-function responses in an acute randomized between-subjects trial against a matched video of the same workout. Flanker and N-back response times were faster (between-group d = 0.37 and 0.48), Flanker P3 latency was shorter (d = 0.41), and accuracy did not improve. In exploratory mediation, the post-exercise systolic blood pressure rise (about 123 to 141 mmHg) mediated the response-time gains, and the lactate rise did not.[5] See Brain.
Brain activity stimulates growth and alteration of tissues such as the muscles; a few days of visualizing unusual postures and movements produced next-day hike-like musclefatigue without physical exercise (trophic nerve effect).[6]
Forty minutes of moderate exercise can drop T3 to zero if glycogen isn't replenished.
— Ray Peat
One of the overlooked things is that muscle activity increases your body temperature and the increased body temperature increases your calorie burning ability. And so it's similar to what thyroid supplementing does. body warming effect of physical activity keeps your metabolic rate going with an anti-inflammatory effect. If you're sedentary, your body's temperature falls and you go into a pro-inflammatory state.
A 6% carbohydrate drink during weight lifting blunted the cortisol rise compared with a non-caloric placebo, and over twelve weeks of progressive training it was linked to larger type I and type II muscle-fibre area gains.[12]
Vigorous exercise (30 min at ~70% heart-rate reserve) dampened the next salivary cortisol spike to a lab stress test versus easier intensities.[13]
Consistently low energy and sleep disturbances are the primary indicators that a training load has become excessive.[14]
Sleep and MPS: One night of total sleep deprivation cut postprandial skeletal muscleprotein synthesis about 18% the next afternoon in 13 healthy young adults (randomized crossover), raised plasma cortisol about 21%, and lowered plasma testosterone about 24%, without a change in measured markers of protein degradation.[15]
Volume vs. intensity must be managed: moderate-intensity training (60% 1RM) stimulates both muscle quality and quantity, whereas lower intensity (40% 1RM) only increases size[17]
Gym focus needs scrutiny; see Activities for the fuller list
Acute rumination: in a within-subject experiment in major depression (n=24; order randomized), one 30-minute bout at 105% of the lactate threshold lowered self-reported rumination versus seated rest, and in a small EEG subsample it shifted the decoded brain state toward distraction. See Depression and Roadmap/06 - Activities.[20]
Gut bacteria and the drive to run: in a mouse study, chemicals made by certain gut bacteria switched on sensory nerves supplying the gut, and these nerves signalled the brain's reward area to raise dopamine during exercise, which kept the mice running. Wiping out the gut bacteria with antibiotics cut treadmill and wheel running by about half, and germ-free mice ran similarly poorly; muscle function was largely unchanged, everyday moving around stayed normal, and running recovered once the antibiotics stopped. Feeding the missing gut chemicals, or giving a drug that acts like dopamine, restored running. Limits: no people were studied, germ-free mice and mice on broad antibiotics are extreme cases, and the experiments were neither randomized nor blinded.[21]
Ammonia toxicity: during exercise, ammonia can double, inhibiting mitochondrial energy production. High-proteinmeals (e.g., 60g) significantly increase ammonia levels in the blood and breath compared to 30g meals.[22]
CO2 and recovery: in rats, transcutaneous carbon dioxide application increased endurance performance, mitochondrial DNA content, and capillary density.[24] A separate hydrogel-based transcutaneous CO2 trial has shown accelerated fracture repair.[25]
Train like a sprinter: short sprints are probably fine, while endurance exercise is the worst choice[26][27]
Lean protein after lifting raises muscleprotein synthesis more than high-fat protein; lipid-rich meat blunted the post-exercise response in a controlled pork comparison.[29]
Don't under-eat protein: methionine-restricted diets (and elevated FGF21) are linked to smaller musculoskeletal development in animal studies, eat enough meat and dairy to avoid this[30]
Physical exercise also supports hippocampal-dependent cognition with aging by restoring adult hippocampal neurogenesis (vascular niche remodeling; neurotrophic and antioxidant factors that reactivate neural stem cells); see Aging.[33]
Optimizing the Anabolic State:
T3: Can maximally stimulate protein synthesis in skeletal muscle at both transcriptional and translational levels (increasing initiation and probably elongation rates) even in the complete absence of measurable circulating insulin or growth hormone.[34]
Young hardgainers are often hypothyroid, impairing ATP and the ability to retain magnesium; supplementing T3 alongside it fixes this.[35]
Hardgainers are often hypothyroid: a low thyroid raises the caloric requirement just to maintain weight, so correcting thyroid function first can matter more than eating more.
I've known a few men in their 20's to early 40's... they would weigh maybe 140 pounds and be five, eight... And no matter how much they ate, they couldn't gain any weight. I suggested they try thyroid, and every one of them immediately started building muscle and growing.
I often ate several thousand calories per day without getting fat, and small noises would shock me awake. Taking thyroid reduced my caloric requirement, and immediately allowed me to sleep deeply. Deficiencies of magnesium, vitamin A, and selenium probably contribute to that metabolic pattern.
Beyond correcting thyroid, the same pro-metabolic foods used elsewhere on this page work in a surplus instead of a deficit: milk and orange juice sipped throughout the day, gelatin alongside muscle meats, and frequent small meals rather than a few large ones. Zinc (as in oysters) and the B-complex vitamins and potassium in brewer's yeast are sometimes worth adding on top of that, though it isn't a calorie-dense food itself.
Supports ATP production, improves strength and lean mass, buffers lactic acid, reduces myostatin (inhibits muscle growth). Improves maximal strength by 5-15%. Increases muscle size through multiple mechanisms. Enhances glycogen storage by ~82% when combined with carbohydrates. Supports cognitive function. Pairing with ~1g sugar/kg bodyweight during loading boosted total musclecreatine retention by about 60% over creatine alone.[45] In 64 sedentary adults aged 45–65 on resistance training plus a 300–500 kcal/day deficit, creatine raised DXA lean tissue by 1.27 kg versus 0.14 kg on placebo over 12 weeks and lowered body fat by 3.24 versus 1.87 percentage points (RCT, exercise arm self-selected); DXA lean tissue includes water, and body water was not measured.[46] Dose: 5g daily
HMB
Anti-catabolic leucine metabolite that reduces muscle breakdown, supports recovery, preserves lean mass during caloric deficits or stress, improves nitrogen retention, supports mitochondrial function
Hard training can suppress the menstrual cycle. Progesterone can become chronically deficient in women who exercise hard, showing up as scant or prolonged menstruation, or amenorrhea. Since progesterone converts into cortisone to meet the added stress of training, well-trained athletes, who need a lot of cortisone, often miss periods for exactly that reason.[54]
In male and female athletes who become infertile from training, progesterone (or its precursor pregnenolone) combined with thyroid support is a reasonable therapy.[55]
Any stress, including heavy exercise, triggers a protective slowing of the thyroid. The slow heart rate often seen in trained runners is largely this adaptive hypothyroidism, not necessarily a sign of superior fitness.[56]
Track temperature and pulse alongside the menstrual cycle itself as a feedback signal, and back off training or add calories if periods become irregular or stop.
A woman's body fat produces hormones that are important for fertility; 30% of body weight being fat can be favorable for health, with 25% probably being the minimum that is safe.
From a bioenergetic perspective, weight loss is not merely a matter of calorie expenditure (CICO), but a result of maintaining a high metabolic rate and minimizing chronic stress.
The modern obesity epidemic is heavily linked to the 300% increase in polyunsaturated fatty acid (PUFA) consumption, along with high iron and high-fructosecorn syrup, rather than refined sugars alone. Studies show that even when controlling for calories, people in high-PUFA environments are significantly heavier than those from previous decades.
Thiamine: There is a strong correlation between obesity and thiamine deficiency; reported deficiency rates range from about 16% to 47% of bariatric surgery candidates.[57]Thiamine is essential for carbon dioxide (CO2) production and ATP synthesis.
Estrogen: This hormone promotes fat storage and can increase the uptake of harmful endotoxins (LPS) from the gut.
Gut clearance: the liver links glucuronic acid to estrogens and excretes them in bile, and some bacterial β-glucuronidase enzymes in the colon strip it off again, so the freed estrone and estradiol can be reabsorbed (in vitro enzyme study).[61] In healthy men, oxytetracycline at 1 g/day for 5 days raised fecal conjugated estrogens and lowered urinary estrogens, while plasma estrone, estradiol and testosterone did not change significantly (small intervention study).[62] In 25 men and 7 postmenopausal women, urinary estrogens rose with fecal microbial diversity and with Clostridia taxa (R=0.50–0.70); no bacterial phylum, Bacteroidetes included, tracked them, and fecal β-glucuronidase activity correlated only with urinary estrone (R=0.36) (cross-sectional study).[63]
Bowel transit: in 40 premenopausal women randomized to senna, wheat bran or loperamide, speeding transit with senna for two menstrual cycles lowered serum estrone and estrone sulfate, wheat bran (about 20 g/day) lowered estrone sulfate, and slowing transit with loperamide caused no significant change (randomized trial).[64] Ten vegetarian women passed heavier stools and more fecal estrogen than 10 omnivores and had lower fecal β-glucuronidase activity; among the vegetarians, plasma estrone and estradiol fell as fecal estrogen output rose (small observational study).[65]
Endorphins: Often praised in exercise, endorphins are actually heavily associated with obesity and can inhibit protective hormones like progesterone and testosterone.[66]
Total daily energy expenditure does not keep climbing with more activity: in 332 adults across five populations, doubly labeled water TEE plateaued at higher accelerometer activity after body-size adjustment (constrained TEE), so stacking endurance miles is a poor bet for raising daily burn.[67]
Intramyocellular lipid: both sedentary/diabetic people and highly-trained endurance athletes can accumulate high muscle fat content, but only in the former is it linked to insulin resistance; in trained athletes it coexists with high insulin sensitivity (the "athlete's paradox"), so this marker alone doesn't indicate a metabolic problem.[68]
Endotoxemia and leaky gut: Strenuous or prolonged exercise increases intestinal permeability, allowing bacteria-derived endotoxins to enter the blood, which triggers inflammation and further promotes fat storage.[69]
In healthy volunteers, even a short bout of maximal aerobic exercise raised bloodendotoxin, and 1,000 mg of vitamin C taken beforehand prevented the rise.[70]
Endurance mileage and androgens: In 15 previously sedentary men who built running to about 56 km/week over 6 months, total testosterone and free androgen index fell significantly; PRL and cortisol also fell, while single-sample LH/FSH and LH pulse frequency (n=5) did not change, and weight loss did not correlate with the testosterone drop.[71]
Always balance stress with nourishment. Restrict calories without slowing metabolism, and avoid feeling hungry, day or night.
If fewer calories yield no weight loss (and it isn't fasting damage), intensify Peating to address root causes (estrogen, stress, gut issues, thyroid) rather than cutting further. Prioritize body repair over weight loss; natural loss likely follows. Temporary weight gain is possible during this process.[72]
Older textbook rules treated intake below about 1,700 kcal/day as enough for anyone to lose weight. In closed metabolic wards, some women who had dieted chronically still maintained weight on about 700 kcal/day; adaptations include a slower thyroid and stress-related muscle atrophy.[73][74]
Among obese patients a group of "efficient metabolisers" can hold excess weight on intakes occasionally as low as about 600 kcal/day; among anorexic women a few fail to reach normal weight on about 3,500 kcal/day and may develop T3 thyrotoxicosis.[75] On a ~1.34 MJ/day (~320 kcal/day) formula diet, efficient metabolisers slowed weight loss as resting metabolic rate, serum T3, and lymphocyte T3 receptor number fell; adding 60 µg/day T3 produced significantly greater weight loss by week 12.[76] Compared with 780 kcal/day, 330 kcal/day for 8 weeks in hospitalized obese subjects blunted the thermic response to food and dropped free T3 faster while resting oxygen uptake fell in both groups.[77]
"The ratio of calcium to phosphate is very important; that's why milk and cheese are so valuable for weight loss, or for preventing weight gain. For people who aren't very active, low fat milk and cheese are better, because the extra fat calories aren't needed."
Eat enough to feel satisfied, not stuffed. Weight loss needs a calorie deficit relative to a raised metabolic rate, not just lower intake; if normally overeating, restrict slightly by lowering fat and maybe starch first.
Pro metabolic, easy to digest, low PUFA, smaller more frequent meals to keep the metabolism up.
Use food to recover after training instead of relying only on willpower.[80]
Maintain high Ca:P ratio. High dairy intake can speed up fat loss even at the same calorie intake: a high-dairy diet increased weight loss by 70% and fat loss by 64% versus a low-calciumdiet at the same calorie deficit.[81] This can allow maintenance calories to rise from around 1500 to 2000.[82] It increases whole-body fat oxidation after meals and suppresses PTH, enhancing mitochondrial uncoupling and T4-to-T3 conversion; evidence for a direct thermogenic (metabolic-rate) boost is weaker and less consistent than the fat-oxidation effect.[83] A chronic diet high in dairy calcium increases whole-body fat oxidation from a meal specifically, and increases in fastingparathyroid hormone relate to decreases in that effect.[84]
High phosphate (grains, muscle meats, excess dairy) causes calcification, weak bones, elevated PTH/FGF23/IL-6, slow metabolism, and fat gain, even with calcium buffering.
Walking uphill/stairs burns meaningfully more per minute than flat walking, with less stress than running, no equipment, and no excuse not to; whether it beats running depends on the pace you'd otherwise run at.[85]
Strength training as the main stressor; add manageable cardio (e.g. 30-min walks) if desired, or any movement you find fun, including swimming
Belly fat and insulin: in a small single-group trial in older men with type 2 diabetes, supervised strength training twice a week for 16 weeks, without a weight-loss diet, raised insulin sensitivity by about 46% and shrank both visceral and subcutaneous belly fat while body weight stayed the same.[86]
Get sunlight, good sleep, and spend time with supportive people; vitamin D via sun/tanning is required, not optional.[87]
Use stress wisely: avoid piling on unnecessary stress (long fasts, excessive cardio, under-eating) beyond training itself
Train sustainably; increase physical activity outside the gym too, and raise volume/intensity gradually rather than all at once
Don't lose fat too quickly. Rapid loss releases stored toxins via free fatty acids, overloading the liver and risking a metabolic crash that can cause diabetes and liver disease.[88] On a 500 or 600 calorie diet with adequate minerals, almost all the weight lost was fat, while a complete fast is very destructive.[89] With a well-fed liver (80 to 100 grams of protein a day) and good thyroid function, the liver can dispose of released PUFA as a toxin without having to oxidize it.[90] Low-fat milk and orange juice, the core of the therapeutic diet, also suit a weight-loss diet.[91] Aim for 6 to 12+ months. Use how you feel and the mirror as guides rather than daily weigh-ins.
Very-low-calorie formula feeding lowers RMR, serum T3, and lymphocyte T3 receptor capacity in "efficient metabolisers"; exogenous T3 can reopen weight loss in that setting, which is not a reason to crash-diet without thyroid and fuel support.[76]
Let's say you were about 50 or 60 pounds overweight, what would you do to lose weight? Go on a low-fat milk and cheese diet, with a little bit of orange juice and an occasional egg and oyster. Calcium and vitamin D are supportive of the thyroid hormone.
1,000 calories a day while still getting all the RDA protein, vitamins and minerals isn't good for your health if you have to lose weight that way, but it's an effective short-term option. Keeping the minerals up is very important. Calcium, magnesium, potassium, and sodium are very protective against too much of the stress reaction.
What should be the focus if a person is gaining weight? Not to eat too many calories first, and then to gradually shift towards a lower fat intake, whatever keeps your temperature up without having an excess of calories.
Sucrose itself, some experiments show that you can increase the metabolic rate by 20% just with sucrose. And so when you do it just right, sugar is a very powerful weight loss instrument.
As your ability to burn fat decreases with your falling thyroid, the cortisol eats up your skeletal muscles that burn fat, and so the unburned fat gets laid down in your trunk and neck and face area.
I saw people lose weight stably when they had the habit of eating large amounts of tortilla chips fried in coconut oil, but those chips disappeared when their producers were pressured into switching to other oils.
If a person really wants to concentrate on losing weight, using milk as their main protein: milk and gelatin are the most favorable for weight loss, partly because the milk comes with such a generous supply of calcium, and calcium powerfully stimulates the metabolic rate.
Q: What about weight loss for women in menopause, if they lose that weight do they then poison themselves with estrogen as it comes out of the cells? Ray Peat: Just temporarily, but it's better to get rid of it and have it local, because inside the cells it produces things such as breast cancer, lung cancer, uterine cancer.
So even as a matter of weight gain, the worst fats for gaining weight are the polyunsaturated vegetable oil fats. The saturated fats don't stimulate weight gain to the same degree.
One thing that often surprises people who want to lose weight is that milk drinkers as a group are the least likely to be fat... Calcium itself is a major factor in preventing the inflammation process which is important in turning on the fat storage business.
The starch isn't quite as stimulating to the metabolic rate as sugar. So the things that I chronically mention in connection with a therapeutic diet happen to be ideal for a weight-loss diet: low-fat milk and orange juice.
On a water fast for 10 days, they found that 80% or more of their weight loss came from destruction of their muscle and skin mass. But if they had a 500-600 calorie diet with adequate minerals, almost all of the weight loss was in the form of fat loss rather than muscle.
[My weight] had been steady at 185 pounds for 25 years, but over a period of six months it dropped to about 175 pounds. I found that eating more coconut oil lowered my weight another few pounds, and eating less caused it to increase.
An increased intake of calcium is the single most important nutritional thing for losing weight. Low-fat milk and cheese should be the main foods; a carrot salad helps to reduce stress hormones.
Cellulose can accelerate transit through the intestine and lower estrogen systemically (partly by simply preventing the reabsorption of estrogen that has been secreted by the bile), but the lignans found in many seeds and grains tend to promote inflammation. Raw carrots, for example, lower estrogen, while flax meal can increase it.
If you lose too fast, that is going to cause damage by oxidative decomposition of the unsaturated fats as they're being oxidized at a higher rate. So I think slow weight loss is the best thing. And having enough protein and minerals in your diet during weight loss can make a tremendous difference in the amount of stress.
[Haidut] Bodybuilders abuse steroids to lose weight and gain lean mass. Considering that testosterone, DHT, and progesterone are anti-lipolytic, what mechanism lets them cause rapid fat loss without really increasing fat oxidation? Ray Peat: The liver, when it's well nourished and activated by thyroid rather than estrogen effects, can harmlessly excrete PUFA. Any PUFA that appears in your bloodstream can be recognized by the liver as a toxin and inactivated by attaching glucuronic acid to it, causing it to be excreted in the urine. [Haidut] So the androgens are basically protecting the liver and allowing it to excrete more PUFA? Ray Peat: Yeah, and thyroid and progesterone and aspirin work in that direction.
Many diet studies have found that you oxidize fat at a high rate the first in some people the first 12 hours and some people the first 24 hours ... at some point you have depleted your stored glycogen and that blocks the formation of the active T3 thyroid hormone and from that point on you're relying for energy to an increased extent on the breakdown of tissue protein turning it into fat and so a more or less starvation diet that goes on for a week or two will I think it's about 80% of your weight loss is in the form of muscle loss and very little maybe 20% in the form of fat loss but if you eat enough sugar and have the minerals such as in fruit and keep a slight protein intake the small amount of sugar will prevent the breakdown of protein tissues and a 10 to 14 day diet in this case will cause you to lose maybe 80% fat and only 20% muscle.
In one experiment people were put on a treadmill ... so that they kept a steady heart rate of no faster than 120 beats per minute just like you were walking down the street and they had blood tests at intervals and almost everyone stopped producing T3 in the first hour of that mild exercise so when your T3 is turned off whether it's by starvation or by excess activity that's the point of which you start breaking down protein tissues.
The carbohydrate at the right amount will spare the protein. So 80 grams of protein a day, for example, if you're getting the carbohydrate from orange juice, comes with minerals. Potassium has a sparing effect on the protein besides the sugar. So fruit juice plus the adequate amount of protein will keep your metabolism up and tend to promote fat loss rather than protein loss.
↑Tarpenning KM, Wiswell RA, Hawkins SA, Marcell TJ. "Influence of weight training exercise and modification of hormonal response on skeletal muscle growth," J Sci Med Sport. 2001;4(4):431-446. PMID 11905937. doi:10.1016/s1440-2440(01)80052-9. Acute session: 7 young men, 3 sets of 10 at 75% 1RM on nine stations after a 4-hour fast; cortisol rose 7% with the 6% carbohydrate drink versus 99% with placebo, while blood glucose and insulin rose. Twelve-week progressive training: carbohydrate during sessions kept blunting cortisol; type I fibre area increased 19.1% and type II 22.5% versus training alone; the cortisol-response difference accounted for 74% (r = 0.8579, p = 0.006) of type I and 52.3% (r = 0.7231, p = 0.043) of type II fibre-area variance.
↑Haycock, B. (2000, October). Hypertrophy-Specific Training. ThinkMuscle Newsletter.
↑Otsuka Y, et al. "Effects of resistance training intensity on muscle quantity/quality in middle-aged and older people: a randomized controlled trial," Journal of Cachexia, Sarcopenia and Muscle. 2022 Apr. https://pubmed.ncbi.nlm.nih.gov/35187867/
↑Spacek LA, et al. "Repeated Measures of Blood and Breath Ammonia in Response to Control, Moderate and High Protein Dose in Healthy Men," Scientific Reports. 2018. https://www.nature.com/articles/s41598-018-20503-0
↑Niikura T, et al. "Topical cutaneous application of carbon dioxide via a hydrogel for improved fracture repair: results of phase I clinical safety trial," BMC Musculoskeletal Disorders. 2019. https://link.springer.com/article/10.1186/s12891-019-2911-7
↑Serra-Prat M, et al. "Intracellular Water Content in Lean Mass as an Indicator of Muscle Quality in an Older Obese Population," Journal of Clinical Medicine. 2020. https://www.mdpi.com/2077-0383/9/5/1580
↑Farmand S, Du Preez A, Kim C, et al. "Exercise counteracts age-associated," Ageing Res Rev. 2025;107:102725. doi:10.1016/j.arr.2025.102725. PMID 40064399.
↑Green AL, et al., "Carbohydrate ingestion augments skeletal muscle creatine accumulation during creatine supplementation in humans," Am J Physiol. 1996;271(5):E821-826.
↑Moretti C, et al. "Pyridoxine (B6) suppresses the rise in prolactin and increases the rise in growth hormone induced by exercise," New England Journal of Medicine. 1982 Aug. https://pubmed.ncbi.nlm.nih.gov/7088124/
↑Karp JR, et al. "Chocolate milk as a post-exercise recovery aid," International Journal of Sport Nutrition and Exercise Metabolism. 2006 Feb. https://pubmed.ncbi.nlm.nih.gov/16676705/
↑Amiri M, et al. "Chocolate milk for recovery from exercise: a systematic review and meta-analysis of controlled clinical trials," European Journal of Clinical Nutrition. 2019 Jun. https://pubmed.ncbi.nlm.nih.gov/29921963/
↑Pritchett K, et al. "Acute effects of chocolate milk and a commercial recovery beverage on postexercise recovery indices and endurance cycling performance," Applied Physiology, Nutrition, and Metabolism. 2009 Dec. https://pubmed.ncbi.nlm.nih.gov/20029509/
↑Gilson SF, et al. "Effects of chocolate milk consumption on markers of muscle recovery following soccer training," Journal of the International Society of Sports Nutrition. 2010 May. https://pubmed.ncbi.nlm.nih.gov/20482784/
↑Papacosta E, et al. "Effects of acute postexercise chocolate milk consumption during intensive judo training on recovery," Applied Physiology, Nutrition, and Metabolism. 2015 Nov. https://pubmed.ncbi.nlm.nih.gov/26513005/
↑Maguire D, et al. "The role of thiamine dependent enzymes in obesity and obesity related chronic disease states: A systematic review," Clinical Nutrition ESPEN. 2018 Jun. https://pubmed.ncbi.nlm.nih.gov/29779823/
↑Tinkov AA, et al. "Influence of iron and copper consumption on weight gain and oxidative stress in adipose tissue of Wistar rats," Interdisciplinary Toxicology. 2012 Sep. https://pubmed.ncbi.nlm.nih.gov/23554552/
↑Dallman MF, et al., "Chronic stress and obesity: a new view of comfort food," Proc Natl Acad Sci USA. 2003;100(20):11696-11701.
↑McCarty MF, Thomas CA. "PTH excess may promote weight gain by impeding catecholamine-induced lipolysis," Medical Hypotheses. 2003 Nov-Dec. https://pubmed.ncbi.nlm.nih.gov/14592784/
↑Goodpaster BH, et al., "Skeletal Muscle Lipid Content and Insulin Resistance: Evidence for a Paradox in Endurance-Trained Athletes," J Clin Endocrinol Metab. 2001;86(12):5755-5761.
↑Ribeiro FM, et al. "Is There an Exercise-Intensity Threshold Capable of Avoiding the Leaky Gut?" Frontiers in Nutrition. 2021. https://pubmed.ncbi.nlm.nih.gov/33763441/
↑Soares MJ, et al., "Postprandial Energy Metabolism in the Regulation of Body Weight: Is there a Mechanistic Role for Dietary Calcium?" Nutrients. 2010;2(6):586-598.
↑Gunther CW, et al. "Fat oxidation and its relation to serum parathyroid hormone in young women enrolled in a 1-y dairy calcium intervention," Am J Clin Nutr. 2005. doi:10.1093/ajcn/82.6.1228.
↑Ibañez J, Izquierdo M, Argüelles I, Forga L, Larrión JL, García-Unciti M, Idoate F, Gorostiaga EM. "Twice-weekly progressive resistance training decreases abdominal fat and improves insulin sensitivity in older men with type 2 diabetes," Diabetes Care. 2005;28(3):662-667. PMID 15735205. Single-group trial with a 4-week pre-training control period; 9 men aged about 67; insulin sensitivity by minimal model rose 46.3% (P < 0.01); visceral fat fell 10.3% and subcutaneous abdominal fat 11.2% by CT (both P < 0.01); fasting glucose fell 7.1%; energy intake rose 15.5%.
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